Our Wellness, etc. Medical Research Team have studied the effects of caffeine on human collagen production for over 30 years. The findings can be summed up in a single concept: "Caffeine inhibits collagen biosynthesis in a dose-dependent manner". Per our research, those individuals suffering from Irritable Bowel Syndrome (IBS), Crohn's and Colitis suffer a progressively more serious condition primarily based on the volume and duration of their caffeine consumption. There are other causes of these conditions, however, caffeine is a primary causation in our research cases.
Is there collagen in our intestines?
The collagen content and the relative amount of collagen types were quantitated in control intestine as well as in both inflamed and strictured intestine resected from patients with Crohn's disease. The major collagen type in control intestine was Type I (68%), followed by Types III (20%) and V (12%).
Does intestinal submucosa contain collagen?
In normal tissue the submucosal layer consist almost entirely of collagen, which is called the skeleton of the small intestine, and it is well known that the fibers run in a criss-cross pattern with a 30 degree angle in the longitudinal direction.
Collagen is the most common protein in the human body and all 28 Types of collagen are critical for good health. Understand that once your collagen production has been inhibited, those cells created with low collagen content are less viable or totally non-viable. In response to detecting these non-viable cells, your immune system attempts to eliminate the cells in a constant cycle of cellular replacement. Every new iteration of cells are compromised or non-viable due to the continued lack of collagen so medical science labels this an autoimmune condition and provides no solution. It should be noted that the energy necessary for this non-stop autoimmune cycle is radical which results in constant state of fatigue for most people who suffer from autoimmune conditions.
Our Wellness, etc. protocols have been shown to increase collagen production by factors. Also, our protocols stabilize and lengthen telomeres in intestinal tissue cells so effectively that the population of healthy, high-integrity RNA/DNA cells completely overrun the small populations of non-viable or mutated cells – typically presenting as lesions.
